Doug
Everytime i get a gun in my hand it...
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- Joined
- Jun 16, 2018
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This is the ULTIMATE-BRUTAL ESTROGENPILL
Testosterone alone DOES NOT fuse your growth plates! Thats just a jewish lie to Keep you away from getting test in your Young prime-puberty years so that you wont become a masculine un-controllable man
"A study was done, giving children 12 years of age Letrozole (Non-Suicidal inhibitor) pills every day through puberty (until 16.)
When they finished, the results were all over the place.
Some kids grew 8 inches, some kids grew 14 inches, some kids stopped growing at 13.
Because of this, the scientists had to put the average height increase at 2".
Why did this happen?
The kids who grew 14 inches were the ones who took their non-suicidal AI religiously.
The kids who grew no inches or two inches were the ones who took their AI for a bit then forgot to take it for two days (huge amounts of estrogen released, growth plates fused)"
What does this tell us?
Aromatose inhibitors are FUCKING GOOD. Kids using medication meant to stop growth were actually growing from it (assuming they didn't forget to take it for a couple of days.)
If you had gotten AI's permanently since the age of 12 until now, your growth plates would be still open, you could theoretically still grow up into your 30s
The average height for men with Estrogen receptor deficiency lies between 200-210cm
If your parents would not had been brainwashed by feministic Propaganda then today you would be a NBA Tier gigachad
Estrogen is the toxic Hormone which causes feminized-stunted manlets and tall masculine women
and If this wouldnt be worse enough, excessive Estrogen along with low T is also responsible for stunting your jaw growth
And yes penis size is also strongly correlated with androgen to Estrogen ratio
This is what our modern world has provided for us, a stunted height/frame along with small genitals and non-dimorphic facial growth
You have been fooled into avoiding red meats and eggs, while consuming ridiculous amounts of pesticide rotten vegetables and calorie-rich-nutritionally-poor-carbs out of plastic packages, just to store a shit amount of fat and Estrogens which have totally prevented you from reaching your true potential
While every 13 year old foid today gets the anti Baby pill prescribed which contains lots of Estrogens which will convert into testosterone in their bodies which will block further Estrogen and make her grow ridiculous tall and thick
Testosterone alone DOES NOT fuse your growth plates! Thats just a jewish lie to Keep you away from getting test in your Young prime-puberty years so that you wont become a masculine un-controllable man
"A study was done, giving children 12 years of age Letrozole (Non-Suicidal inhibitor) pills every day through puberty (until 16.)
When they finished, the results were all over the place.
Some kids grew 8 inches, some kids grew 14 inches, some kids stopped growing at 13.
Because of this, the scientists had to put the average height increase at 2".
Why did this happen?
The kids who grew 14 inches were the ones who took their non-suicidal AI religiously.
The kids who grew no inches or two inches were the ones who took their AI for a bit then forgot to take it for two days (huge amounts of estrogen released, growth plates fused)"
What does this tell us?
Aromatose inhibitors are FUCKING GOOD. Kids using medication meant to stop growth were actually growing from it (assuming they didn't forget to take it for a couple of days.)
If you had gotten AI's permanently since the age of 12 until now, your growth plates would be still open, you could theoretically still grow up into your 30s
The average height for men with Estrogen receptor deficiency lies between 200-210cm
If your parents would not had been brainwashed by feministic Propaganda then today you would be a NBA Tier gigachad
Estrogen is the toxic Hormone which causes feminized-stunted manlets and tall masculine women
and If this wouldnt be worse enough, excessive Estrogen along with low T is also responsible for stunting your jaw growth
INTRODUCTION:
Sagittal jaw growth is influenced during puberty by a ratio of androgens and estrogens.
The CYP19A1 (formerly CYP19) gene encodes the cytochrome P450 enzyme aromatase (estrogen synthetase), which converts testosterone to estrogen. Genetic variations including single nucleotide polymorphisms might regulate CYP19A1 gene expression or the function of the aromatase protein and thus influence sagittal jaw growth.
METHODS:
The annual sagittal jaw growth in 92 pubertal orthodontic patients was determined by using pretreatment and posttreatment cephalometric radiographs. Single nucleotide polymorphisms rs2470144 and rs2445761 were genotyped and haplotypes constructed. Associations between genotypes or haplotypes and the annual sagittal growth were estimated by using JMP (version 9.0; SAS Institute, Cary, NC).
RESULTS:
Two single nucleotide polymorphisms were significantly associated with average differences in annual sagittal jaw growth in boys. Haplotype analysis demonstrated that haplotypes T(rs2470144)T(rs2445761) and C(rs2470144)T(rs2445761) had significant effects on annual sagittal maxillary growth and on mandibular growth in boys. No association was found in girls.
CONCLUSIONS:
A quantitative trait locus that influences male pubertal sagittal jaw growth might exist in the CYP19A1 gene, and single nucleotide polymorphisms rs2470144 and rs2445761 might be inside this quantitative trait locus or be linked to it.
Sagittal jaw growth is influenced during puberty by a ratio of androgens and estrogens.
The CYP19A1 (formerly CYP19) gene encodes the cytochrome P450 enzyme aromatase (estrogen synthetase), which converts testosterone to estrogen. Genetic variations including single nucleotide polymorphisms might regulate CYP19A1 gene expression or the function of the aromatase protein and thus influence sagittal jaw growth.
METHODS:
The annual sagittal jaw growth in 92 pubertal orthodontic patients was determined by using pretreatment and posttreatment cephalometric radiographs. Single nucleotide polymorphisms rs2470144 and rs2445761 were genotyped and haplotypes constructed. Associations between genotypes or haplotypes and the annual sagittal growth were estimated by using JMP (version 9.0; SAS Institute, Cary, NC).
RESULTS:
Two single nucleotide polymorphisms were significantly associated with average differences in annual sagittal jaw growth in boys. Haplotype analysis demonstrated that haplotypes T(rs2470144)T(rs2445761) and C(rs2470144)T(rs2445761) had significant effects on annual sagittal maxillary growth and on mandibular growth in boys. No association was found in girls.
CONCLUSIONS:
A quantitative trait locus that influences male pubertal sagittal jaw growth might exist in the CYP19A1 gene, and single nucleotide polymorphisms rs2470144 and rs2445761 might be inside this quantitative trait locus or be linked to it.
And yes penis size is also strongly correlated with androgen to Estrogen ratio
This is what our modern world has provided for us, a stunted height/frame along with small genitals and non-dimorphic facial growth
You have been fooled into avoiding red meats and eggs, while consuming ridiculous amounts of pesticide rotten vegetables and calorie-rich-nutritionally-poor-carbs out of plastic packages, just to store a shit amount of fat and Estrogens which have totally prevented you from reaching your true potential
While every 13 year old foid today gets the anti Baby pill prescribed which contains lots of Estrogens which will convert into testosterone in their bodies which will block further Estrogen and make her grow ridiculous tall and thick