What makes black men superior?

[MongoloidJoe]

Are they the Supreme Race

 

[PersonalityChad]

No.

 

[lifelongkhhv]

They really are not superior but they are more resistant to problems caused by strong sunlight, apart from that being black has only disadvantages

 

[Lonelyus]

The warrior gene, The recent social support, Uzis, Mac 10, Ak47

 

[asianfuck]

Are they the Supreme Race

 

[BataviaCel]

NIGGERS!!!

 

[Luzifer]

Are they the Supreme Race

What drugs r u using

 

[NothinButAComedy]

Race is a social construct

 

[Murder Mario]

Race is a social construct

 

[NothinButAComedy]

People use this so much it’s lost meaning

 

[Murder Mario]

People use this so much it’s lost meaning

calling something that is based in reality a social construct has been used by left wing cucks so much it’s lost meaning.

 

[Serpents reign]

Are they the Supreme Race

View: https://www.bitchute.com/video/geKov03ufzLU/

 

[MongoloidJoe]

View: https://www.bitchute.com/video/geKov03ufzLU/

Violence is based

 

[Serpents reign]

Race is a social construct

Just like gender and Health concerns like diabetes and obesity and cancer and aids and mortality.

 

[MongoloidJoe]

The warrior gene, The recent social support, Uzis, Mac 10, Ak47

 

[Uninvited]

They are good for working fields in the sun, as long as you can keep them in line.
For everything else history has shown that they are useless

 

[8CbZA-kHFu6pFgE*]

They may be physically superior to other races but in terms of intelligence they are the least intelligent race by far. Otherwise Sub Saharan African countries would have had meaningful colonies and not be a complete shithole.

 

[Youthpilled]

Username checks out, if you actually believe that niggers are superior. Black men are, quite frankly, lackluster in every regard, except for sports. Even in terms of SMV, the BBCpill has been proven to be highly exaggerated and even when BMWF couples emerge, it's always the pairing between legitimate Tyrone and some ghetto trash lardwhale.

 

[CrackingYs]

They aren't superior in any way except certain types of athleticism and it comes at a big disadvantage. Their brains are like 40% smaller than human brains. They are more likely related to Homoerectus and/or Neanderthal (extinct non-humans), and not Homosapiens (human). Therefore they may not actually be human beings to begin with. Basically humans used to keep them around as slaves cause that's all they're really good for. It was a massive mistake to give them human rights. Just look at Africa to see how dysfunctional a whole continent of black people is. They're totally doomed, and there's no white people around to blame.

 

[Med Anime]

they can't be superior whent they're inside a cage

 

[Drakul]

Also they can't get fully hard and unless Tyrone are ugly almost as the average Indian

 

[FakeFakecel]

View: https://www.bitchute.com/video/geKov03ufzLU/

Wtf

 

[futureprettyboy]

OP is a cumbrain, too deranged from watching porn.

 

[Drakul]

Dick size nothing else, if i see anyone dating a black man i auto assume its because of dick

Debunked. Blacks aren't bigger than whites on average they're just selected more because Jews want you to feel cucked

 

[Drakul]

Source?

There's no source providing data about blacks being significantly bigger than whites

 

[Intellau_Celistic]

Blacks have the variants of HMGA2 and KIBRA for superior intelligence.

1000Genomes_30x	Global	Study-wide	6404	C=0.4775	T=0.5225
1000Genomes_30x	African	Sub	1786	C=0.3740	T=0.6260
1000Genomes_30x	Europe	Sub	1266	C=0.6509	T=0.3491
1000Genomes_30x	South Asian	Sub	1202	C=0.7063	T=0.2937

When the scientists zeroed in on the DNA of people whose images showed smaller brains, they found a consistent relationship between subtle shifts in the genetic code and diminished memory centers. Furthermore, the same genes affected the brain in the same ways in people across diverse populations from Australia, North America and Europe, suggesting new molecular targets for drug development.


“Millions of people carry variations in their DNA that help boost or lower their brains’ susceptibility to a vast range of diseases,” said Thompson. “Once we identify the gene, we can target it with a drug to reduce the risk of disease. People also can take preventive steps through exercise, diet and mental stimulation to erase the effects of a bad gene.”


In an intriguing twist, Project ENIGMA investigators also discovered genes that explain individual differences in intelligence. They found that a variant in a gene called HMGA2 affected brain size as well as a person’s intelligence.


DNA is comprised of four bases: A, C, T and G. People whose HMGA2 gene held a letter “C” instead of “T” on that location of the gene possessed larger brains and scored more highly on standardized IQ tests.

An international team of scientists has successfully mapped the human genes that either increase or disrupt the brain's resistance to various neurological and mental illnesses. This study, presented in the journal Nature Genetics, has identified novel genes that could provide insight into the differences that exist in brain size and intelligence. The results could also lead to the development of new drug treatments.

'Our individual centres couldn't review enough brain scans to obtain definitive results,' says Professor Thompson. 'By sharing our data with project ENIGMA, we created a sample large enough to reveal clear patterns in genetic variation and show how these changes physically alter the brain.'

What set this work apart from past research studies was that researchers measured the brain's size and memory centres in many magnetic resonance imaging (MRI) images from more than 21 100 healthy subjects, while screening their deoxyribonucleic acid (DNA) as well.

'Millions of people carry variations in their DNA that help boost or lower their brains' susceptibility to a vast range of diseases,' the UCLA researcher says. 'Once we identify the gene, we can target it with a drug to reduce the risk of disease. People also can take preventive steps through exercise, diet and mental stimulation to erase the effects of a bad gene.'

The team also found genes that explain individual differences in intelligence, uncovering a variant in a gene called HMGA2 that affected both brain size and intelligence. DNA has four bases: A, C, T and G; subjects whose HMGA2 gene had C instead of T had larger brains and recorded higher results on standardised intelligence quotient (IQ) tests.

https://cordis.europa.eu/article/id/34518-connection-found-between-genes-and-brain-size-intelligence

Population	Group	Sample Size	Ref Allele	Alt Allele

Population	Group	Sample Size	Ref Allele	Alt Allele
Total	Global	283370	C=0.479617	T=0.520383
European	Sub	238688	C=0.487787	T=0.512213
African	Sub	11348	C=0.62328	T=0.37672
African Others	Sub	390	C=0.685	T=0.315
African American	Sub	10958	C=0.62110	T=0.37890

 

[EngelCallDell19]

Are they the Supreme Race

idk you tell me? are niggers a supreme race?

 

[MillionBashStream97]

Ask cracker women, and they will tell you.

 

[EngelCallDell19]

Username checks out, if you actually believe that niggers are superior. Black men are, quite frankly, lackluster in every regard, except for sports. Even in terms of SMV, the BBCpill has been proven to be highly exaggerated and even when BMWF couples emerge, it's always the pairing between legitimate Tyrone and some ghetto trash lardwhale.

>say cracker to a white person
>white man won't give a shit
>say nigger to a nigger
>nigger starts seething and goes on a rant about systematic racism and white privilege
niggers wanna talk about white fragility, but the second you say nigger goddamn they get real mad

 

[Intellau_Celistic]

Single nucleotide polymorphisms (SNP) of certain genes become more and more relevant as potential modulators of cognitive function. It has been shown that young
homozygous C-allele carriers of the KIBRA-SNP rs17070145 exhibit a lower memory performance and hippocampal (HC) activity as well as a smaller volume of HC subfields
than T-allele carriers (1, 2). However, a potential impact of the KIBRA-SNP on memory functions and related brain structures, especially during aging, is still a matter of
debate (3). Therefore, here we addressed whether KIBRA-SNP status is linked with memory performance as well as HC volume and microstructural integrity in older
subjects and whether this is modulated by vascular risk factors (4).

KIBRA groups neither differed in demographic parameters as age, sex or
education nor in potential risk factors for age-dependent decline (Tab. 1).
Adjusting for systolic blood pressure did not attenuate the effect that KIBRA T-
allele carriers showed a better memory consolidation (ANCOVA, F (1, 137) = 5.61,
p = 0.019; Fig. 2) and a larger mean HC volume (ANCOVA, F (1, 137) = 6.33, p =
0.013; Fig. 3) than homozygous C-allele carriers. The volumetric differences can
be selectively explained by larger volumes of the cornu ammonis (CA)2/3
(ANCOVA, F (1, 137) = 9.23, p = 0.003; Fig. 3) and the CA4/dentate gyrus (DG)
(ANCOVA, F (1, 137) = 8.81, p = 0.004 ; Fig. 3) subfields. Furthermore, MD (as an
inverse parameter for microstructural integrity) was not different in the total HC
between the KIBRA groups. However, T-allele carriers tended to show a reduced
MD in the HC subfields CA1, CA2/3, CA4/DG and subiculum, reaching
significance in the right CA4/DG subfield independent of the arterial blood
pressure (ANCOVA, F (1, 137) = 10.26, p = 0.002)

The results of this cross-sectional study confirm previous findings (5) of a better memory performance in KIBRA-SNP T-allele carriers in comparison to homozygous C-allele
carriers in a well-characterized sample of older people, independent of the arterial blood pressure, one of the highest risk factors for cognitive decline. Moreover, we could
show for the first time that T-allele carriers exhibited a larger volume and partially better microstructure within those HC subfields that offer neurogenesis and long-term
potentiation, key features linked with memory processes. The identification of neuronal mechanisms and genetic predisposition, underlying cognitive function and synaptic
plasticity can be of clinical benefit to develop new prevention, diagnosis and therapy strategies against age-associated cognitive decline.

https://www.researchgate.net/profil...nd-microstructure-of-healthy-older-adults.pdf

In a sample of 25 pairs of EOS proband-healthy full sibling, we sought to investigate the association of KIBRA with memory performance. Episodic memory was measured using immediate and delayed recall measures of the California Verbal Learning Test. In a combined analysis (TT vs. TC/CC) assuming a C dominant model of inheritance, we found a main effect of genotype where individuals with TT genotype outperformed non-TT-carriers at immediate and delayed recall.

We report a significant association of rs17070145 with both episodic (r = 0.068, P = 0.001) and working memory (r = 0.035, P = 0.018). In summary, our findings indicate that SNP rs17070145 located within KIBRA explains 0.5% of the variance for episodic memory tasks and 0.1% of the variance for working memory tasks in samples of primarily Caucasian background.

https://onlinelibrary.wiley.com/doi/abs/10.1002/ajmg.b.32101

Moreover, among older adults T-allele carriers of the examined KIBRA polymorphism showed better spatial learning compared to C homozygotes. Together these findings provide the first evidence for an effect of the KIBRA rs17070145 polymorphism on spatial memory in humans and age differences in the reliance on landmark and boundary-related spatial information.

Impact of common KIBRA allele on human cognitive functions - PubMed​

The rs17070145 polymorphism (C → T substitution, intron 9) of the KIBRA gene has recently been associated with episodic memory and cognitive flexibility. These findings were inconsistent across reports though, and largely lacked gene-gene or gene-environment interactions. The aim of the present...

pubmed.ncbi.nlm.nih.gov

The rs17070145 polymorphism (C → T substitution, intron 9) of the KIBRA gene has recently been associated with episodic memory and cognitive flexibility. These findings were inconsistent across reports though, and largely lacked gene-gene or gene-environment interactions. The aim of the present study was to determine the impact of the rs17070145 polymorphism on clinically relevant cognitive domains and its interaction with the modifiers 'lifestyle' and 'cardiovascular risk factors'. Five-hundred forty-five elderly volunteers (mean age 64 years, ±7 years, 56% women) accomplished a comprehensive cognitive testing. Principal component analysis was used to reveal the internal structure of the data, rendering four composite scores: verbal memory, word fluency, executive function/psychomotor speed, and working memory. Lifestyle was assessed with a detailed questionnaire, age-associated risk factors by clinical interview and examination. There was no main effect of the rs17070145 genotype on any cognitive composite scores. However, we found worse performance in executive functions for T-allele carriers in the presence of arterial hypertension (β=-0.365, p=0.0077 and 0.031 after Bonferroni correction). This association was further modified by gender, showing the strongest association in hypertensive females (β=-0.500, p=0.0072 and 0.029 after Bonferroni correction). The effect of KIBRA on cognitive function seems to be complex and modified by gender and arterial hypertension.

Posts91,872

• Apr 13, 2023
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• #31

What?

The above is a TT carrier. He is on defense.

ALFA Allele Frequency
The ALFA project provide aggregate allele frequency from dbGaP. More information is available on the project page including descriptions, data access, and terms of use.

Release Version: 20201027095038
Search:

Population	Group	Sample Size	Ref Allele	Alt Allele

Population	Group	Sample Size	Ref Allele	Alt Allele
Total	Global	65622	C=0.61295	T=0.38705
European	Sub	46144	C=0.67573	T=0.32427
African	Sub	5836	C=0.4231	T=0.5769
African Others	Sub	202	C=0.371	T=0.629
African American	Sub	5634	C=0.4249	T=0.5751
Asian	Sub	502	C=0.189	T=0.811
East Asian	Sub	402	C=0.169	T=0.831

The rs17070145-T variant of the WWC1 gene, coding for the KIBRA protein, has been associated with both increased episodic memory performance and lowered risk for late onset Alzheimer's disease, although the mechanism behind this protective effect has not been completely elucidated. To achieve a better understanding of the pathways modulated by rs17070145 and its associated functional variant(s), we used laser capture microdissection (LCM) and RNA-sequencing to investigate the effect of rs17070145 genotypes on whole transcriptome expression in the human hippocampus (HP) of 22 neuropathologically normal individuals, with a specific focus on the dentate gyrus (DG) and at the pyramidal cells (PC) of CA1 and CA3 sub-regions. Differential expression analysis of RNA-seq data within the HP based on the rs17070145 genotype revealed an overexpression of genes involved in the MAPK signaling pathway, potentially driven by the T/T genotype. The most important contribution comes from genes dysregulated within the DG region. Other genes significantly dysregulated, and not involved in the MAPK pathway (Adj P < 0.01 and Fold Change > |1.00|) were: RSPO4 (HP); ARC, DUSP5, DNAJB5, EGR4, PPP1R15A, WBP11P1, EGR1, GADD45B (DG); CH25H, HSPA1A, HSPA1B, TNFSF9, and NPAS4 (PC). Several evidences suggested that the MAPK signaling pathway is linked with memory and learning processes. In non-neuronal cells, the KIBRA protein is phosphorylated by ERK1/2 (involved in the MAPK signaling) in cells as well as in vitro. Several of the other dysregulated genes are involved in memory and learning processes, as well as in Alzheimer's Disease. In conclusion, our results suggest that the effect of the WWC1 rs17070145 polymorphism on memory performance and Alzheimer's disease might be due to a differential regulation of the MAPK signaling, a key pathway involved in memory and learning processes.

When you see an African STEM student, assume he has the TT genotype.

Coupling with any of the SNPs below, for example, provides an IQ advantage over the common European male.

A genome-wide association study (GWAS) of educational attainment was conducted in a discovery sample of 101,069 individuals and a replication sample of 25,490. Three independent single-nucleotide polymorphisms (SNPs) are genome-wide significant (rs9320913, rs11584700, rs4851266), and all three replicate. Estimated effects sizes are small (coefficient of determination R2 ≈ 0.02%), approximately 1 month of schooling per allele. A linear polygenic score from all measured SNPs accounts for ≈2% of the variance in both educational attainment and cognitive function. Genes in the region of the loci have previously been associated with health, cognitive, and central nervous system phenotypes, and bioinformatics analyses suggest the involvement of the anterior caudate nucleus. These findings provide promising candidate SNPs for follow-up work, and our effect size estimates can anchor power analyses in social-science genetics.

https://www.science.org/doi/abs/10.1126/science.1235488

 

[Monke]

Supreme Race

Vous etes rasiste

 

[AsiaCel]

They have good fast twitch muscles for chimping out but they suck at everything else, including warfare.

And that applies exclusively to West Africans who are stronger. East Africans like ethopians and somalis are lightbulb heads who are only good at running

 

[Lonelyus]

Cope they only kill each other

You have that gene too niggah!

 

[Intellau_Celistic]

View attachment 842641View attachment 842642View attachment 842643
idk you tell me? are niggers a supreme race?

The kids appear to have larger heads.

European	Sub	222080	C=0.473032	T=0.526968
African	Sub	9026	C=0.5011	T=0.4989

 

[John Kelly]

Girls like Black Men because some of them are Tall but Black Incels live in Hell

 

[Intellau_Celistic]

Girls like Black Men because some of them are Tall but Black Incels live in Hell

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