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JFL Jewish Intelligence Versus Yellow Peril

Intellau_Celistic

Intellau_Celistic

5'3 KHHV Mentalcel
★★★★★
Joined
Aug 26, 2021
Posts
163,414
In Voltaire's A Philosophical Dictionary, he wrote of Jews: "In short, we find in them only an ignorant and barbarous people, who have long united the most sordid avarice with the most detestable superstition and the most invincible hatred for every people by whom they are tolerated and enriched."[153] Telushkin states that Voltaire did not limit his attack to aspects of Judaism that Christianity used as a foundation, repeatedly making it clear that he despised Jews.[150]

"This is for Gohan!"

The Ashkenazim...a group bestowed with "high IQ"! Their VIQ is quite phenomenal:

One feature that stands out in the Jewish cluster: The We’ve yet to find evidence that Jewish intelligence or philanthropy is genetic.average verbal IQ score among Jews is 120. Translate that into percentiles and we discover that the average Jew is a “one out of ten”—brighter than 91% of the population. That may explain why Jews, at only 1.4% of the American population, make up 22% of Ivy League students, 20% of America’s chief executives and have earned 25% of the Nobel Prizes awarded to American scientists (not including Jews born outside the US, such as Albert Einstein) since 1950—32% worldwide in the 21st century—and 52%(!) of Pulitzer Prizes for nonfiction (the key is verbal IQ).1


https://www.chabad.org/library/article_cdo/aid/498027/jewish/Are-Jews-a-Race.htm


In Commentary in April 2007, Charles Murray, the coauthor of The Bell Curve (1994), again made the claim that “Jews are smarter” than everyone else (Murray, 2007). In The Bell Curve, he and his late (and Jewish) coauthor, Richard Herrnstein, first presented their argument about the intellectual superiority of “Ashkenazic Jews of European origins,” who “test higher than any other ethnic group (Murray and Herrnstein, 1994). Jews in America and Britain have an overall IQ mean somewhere between a half and a full standard deviation above the mean, with the source of the difference concentrated in the verbal component.” Murray again makes the argument of an unmistakable “Jewish Genius,” in Commentary, but now with an explanation:


But is it all in the genes? So far, no real evidence. No “Jewish genes” for IQ scores have turned up, nor for scientific innovation (although over 500 genes have been found related to intelligence, so we may not be far).

Indeed. The research shows something quite different, dear Jew:


1648671725747


(Mine)


Chromo


(Also mine)

We gather more evidence:


In a sample of 25 pairs of EOS proband-healthy full sibling, we sought to investigate the association of KIBRA with memory performance. Episodic memory was measured using immediate and delayed recall measures of the California Verbal Learning Test. In a combined analysis (TT vs. TC/CC) assuming a C dominant model of inheritance, we found a main effect of genotype where individuals with TT genotype outperformed non-TT-carriers at immediate and delayed recall.


A common T/C polymorphism within the ninth intron of the KIBRA gene (rs17070145) is thought to influence memory in humans. Since cognitive impairment, including memory, is a core feature of schizophrenia, we attempted to investigate this association in an independent sample of adolescent...
pubmed.ncbi.nlm.nih.gov


https://www.researchgate.net/profile/Theresa-Koebe-2/publication/266794442_Impact_of_KIBRA-SNP_rs17070145_on_memory_performance_and_hippocampal_volume_and_microstructure_of_healthy_older_adults/links/543bd92c0cf204cab1db413b/Impact-of-KIBRA-SNP-rs17070145-on-memory-performance-and-hippocampal-volume-and-microstructure-of-healthy-older-adults.pdf


1648671818663




We report a significant association of rs17070145 with both episodic (r = 0.068, P = 0.001) and working memory (r = 0.035, P = 0.018). In summary, our findings indicate that SNP rs17070145 located within KIBRA explains 0.5% of the variance for episodic memory tasks and 0.1% of the variance for working memory tasks in samples of primarily Caucasian background.


https://onlinelibrary.wiley.com/doi/abs/10.1002/ajmg.b.32101


Moreover, among older adults T-allele carriers of the examined KIBRA polymorphism showed better spatial learning compared to C homozygotes. Together these findings provide the first evidence for an effect of the KIBRA rs17070145 polymorphism on spatial memory in humans and age differences in the reliance on landmark and boundary-related spatial information.


https://onlinelibrary.wiley.com/doi/abs/10.1002/hipo.22148

A genotype that increases VIQ? And perhaps even PIQ? How common is it among the "Chosen Race"?


Ken



A variant common in East Asians yet noticeably absent among the Jews? And the rate appears to mirror the rate of TT genotype in Europeans. Most interesting for a "Superior" race.

Let us continue:


https://citeseerx.ist.psu.edu/viewdoc/download?doi=10.1.1.997.316&rep=rep1&type=pdf

(Nine cohorts)


Intelligence measure: Participants’ cognitive ability was measured at their intake
assessment, using a short form of the Wechsler Adult Intelligence Scale-Revised (WAIS-R)
for participants age 16 or older, or the Wechsler Intelligence Scale for Children-Revised
(WISC-R) for those younger than 16. The short-form consisted of two Verbal subtests
(information and vocabulary) and two Performance subtests (block design and picture
arrangement), selected because performance on these subtests correlates .90 with overall IQ 30 .
An estimate of full-scale IQ was determined by prorating the scaled scores for these four
subtests


1648672066879


(Mine)


CCCC


(Also mine)


Rs6042314


(rs6042314 is consistently replicated as an IQ gene)

Frequency among the Jews? Well, it seems to be unknown.

However:


Edu



The frequency is quite high among Europeans and East Asians, so surely this gene did not influence the Jewish IQ?

Our last gene:


Although individual differences in intelligence (general cognitive ability) are highly heritable, molecular genetic analyses to date have had limited success in identifying specific loci responsible for its heritability. This study is the first to investigate exome variation in individuals of...
www.nature.com


lthough individual differences in intelligence (general cognitive ability) are highly heritable, molecular genetic analyses to date have had limited success in identifying specific loci responsible for its heritability. This study is the first to investigate exome variation in individuals of extremely high intelligence. Under the quantitative genetic model, sampling from the high extreme of the distribution should provide increased power to detect associations. We therefore performed a case–control association analysis with 1409 individuals drawn from the top 0.0003 (IQ >170) of the population distribution of intelligence and 3253 unselected population-based controls. Our analysis focused on putative functional exonic variants assayed on the Illumina HumanExome BeadChip. We did not observe any individual protein-altering variants that are reproducibly associated with extremely high intelligence and within the entire distribution of intelligence. Moreover, no significant associations were found for multiple rare alleles within individual genes. However, analyses using genome-wide similarity between unrelated individuals (genome-wide complex trait analysis) indicate that the genotyped functional protein-altering variation yields a heritability estimate of 17.4% (s.e. 1.7%) based on a liability model. In addition, investigation of nominally significant associations revealed fewer rare alleles associated with extremely high intelligence than would be expected under the null hypothesis. This observation is consistent with the hypothesis that rare functional alleles are more frequently detrimental than beneficial to intelligence.
Click to expand...


Although individual differences in intelligence (general cognitive ability) are highly heritable, molecular genetic analyses to date have had limited success in identifying specific loci responsible for its heritability. This study is the first to investigate exome variation in individuals of...
www.nature.com


CT



TCC


(My Sample)

Frequency in Jews:


Ash C



Again...quite rare in the Ashkenazim. Quite common in East Asians.
 
Little corroboration of the so-called "Jewish Intellect".

Let us look at the journals:

M1


M2


M3
 
Jews own the Nobel peace prize
Jews own the media
Jews own journalism
Jews own American rap music rock music and even some Jazz:feelsmage::feelsmage::feelsmage::feelsmage::feelsmage::feelsmage:
 
Gigaiq thread
 
Ashkenazi jews have a very high verbal indeed but poor or average visuo-spatial intelligence. No denying that. Ask them to do some origami or fold a pizza box, or do a ravens progressive matrices test and they are like a fish out of water.

And East Asians are the opposite. We have high visuo-spatial compared to our verbal.

In the end, in my opinion, we East Asians are vastly more intelligent than ashkenazi jews. Visuo-spatial intelligence (non-verbal) is objective and performance intelligence and thus weighs much more than verbal intelligence which is subjectivity based.

Of course in western civilization, verbal intelligence is far more valued (to its own detriment). That is why Jews are placed so high and seem so smart. People who are quick mouthed and linear thinking aren't necessarily the smartest. Far from that. Its the opposite actually.

In the end, imo, non-linear thinking and objective non-verbal reasoning wins because it is analaguous to the way the universe works ultimately. Almost akin to believing in god. Objective reasoning is what I try to do all the time here if you guys haven't noticed. I leave myself out of the picture and just explain things the way that they are. Irrespective if it benefits me or not.

Linear thinking and subjective verbal reasoning is only focusing on a narrow aspect. And to me, akin to believing in the devil.
 
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Of course in western civilization, verbal intelligence is far more valued (to its own detriment). That is why Jews are placed so high and seem so smart. People who are quick mouthed and linear thinking aren't necessarily the smartest. Far from that. Its the opposite actually.
High IQ. It's always felt unnatural to me how much the West prizes loud fast talkers over quiet people who deliver results.
 
Ashkenazi jews have a very high verbal indeed but poor or average visuo-spatial intelligence. No denying that. Ask them to do some origami or fold a pizza box, or do a ravens progressive matrices test and they are like a fish out of water.

And East Asians are the opposite. We have high visuo-spatial compared to our verbal.

In the end, in my opinion, we East Asians are vastly more intelligent than ashkenazi jews. Visuo-spatial intelligence (non-verbal) is objective and performance intelligence and thus weighs much more than verbal intelligence which is subjectivity based.

Of course in western civilization, verbal intelligence is far more valued (to its own detriment). That is why Jews are placed so high and seem so smart. People who are quick mouthed and linear thinking aren't necessarily the smartest. Far from that. Its the opposite actually.

In the end, imo, non-linear thinking and objective non-verbal reasoning wins because it is analaguous to the way the universe works ultimately. Almost akin to believing in god. Objective reasoning is what I try to do all the time here if you guys haven't noticed. I leave myself out of the picture and just explain things the way that they are. Irrespective if it benefits me or not.

Linear thinking and subjective verbal reasoning is only focusing on a narrow aspect. And to me, akin to believing in the devil.
Their affinity with and aptitude in verbal intelligence has allowed them to alter the public narrative surrounding themselves and created the mythos of the "Jewish IQ" when in reality they're as average as other races in the aggregate.
 
Mother:

Mother


Myself:

Myself
 
Myself:

hapmap-png.592389


Myself1

(30X Whole Genome Sequencing)


ALFA Allele Frequency
The ALFA project provide aggregate allele frequency from dbGaP. More information is available on the project page including descriptions, data access, and terms of use.

Release Version: 20201027095038
Search:
PopulationGroupSample SizeRef AlleleAlt Allele

PopulationGroupSample SizeRef AlleleAlt Allele
Total Global11572G=0.70930A=0.00000, C=0.29070
European Sub9024G=0.6417A=0.0000, C=0.3583
African Sub2010G=0.9866A=0.0000, C=0.0134
African Others Sub86G=0.99A=0.00, C=0.01
African American Sub1924G=0.9865A=0.0000, C=0.0135
Asian Sub4G=0.0A=0.0, C=1.0
East Asian Sub2G=0.0A=0.0, C=1.0
 
jesus christ ,you're so intelligent intellau ,never know that you can understand this genetic science, did you learned online or what?
 
"this is for gohan"
Gohan GIF - Découvrez sur GIFER
 
@Intellau_Celistic where
 
jesus christ ,you're so intelligent intellau ,never know that you can understand this genetic science, did you learned online or what?

I'm a novice at the moment, sadly. This is only a preliminary review.
 
We conduct a large-scale genetic association analysis of educational attainment in a sample of ~1.1 million individuals and identify 1,271 independent genome-wide-significant SNPs. For the SNPs taken together, we found evidence of heterogeneous effects across environments. The SNPs implicate genes involved in brain-development processes and neuron-to-neuron communication. In a separate analysis of the X chromosome, we identify 10 independent genome-wide-significant SNPs and estimate a SNP heritability of ~0.3% in both men and women, consistent with partial dosage compensation. A joint (multi-phenotype) analysis of educational attainment and three related cognitive phenotypes generates polygenic scores that explain 11–13% of the variance in educational attainment and 7–10% of the variance in cognitive performance. This prediction accuracy substantially increases the utility of polygenic scores as tools in research.


1689792588992

1689793097515


1689793232980

1689793249916


1689793383903

1689793399346

1689793593112

1689793627046

1689793652850


1689793769985

1689793791707

(Mirrors Europeans)

1689793858643
 
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An association study on the polymorphisms of dopaminergic genes with working memory in a healthy Chinese Han population.​

Gong P et al. 2012
A population-based study was performed to examine the effects of COMT, DAT (1), DRD (1), DRD (2), DRD (3), and DRD (4) on cognitive ability in a healthy population. The result indicated there were significant associations of TaqIA and TaqIB in DRD (2) with digital working memory span. Also found was a significant interaction between Ser9Gly in DRD (3) and TaqIA of DRD (2) on digital WM span. However, COMT, DAT (1) , DRD (1), and DRD (4) had no significant effects on digital and spatial WM spans.


1689798135040
 

Evidence for specificity of polygenic contributions to attainment in English, maths and science during adolescence.​

Donati G et al. 2021
Researchers performed genome-wide association studies of standardised national English, maths and science tests using data from the UK-based Avon Longitudinal Study of Parents and Children (ALSPAC) to assess whether there exist academic subject-specific molecular genetic contributions. The performance in the three academic subjects were assessed using National Curriculum-based Standardized Assessment tests (SATs) at 11 and 14 years of age. The academic attainment scores for English (N = 5983), math (N = 6017) and science (N = 6089) were calculated by summing age- and sex-regressed SAT scores from these two time points for each academic subject. After the genotyping, one genome-wide significant single nucleotide polymorphism (SNP) was identified for attainment in science, but none for attainment in English or math. Further 26 independent SNPs showed suggestive evidence of association with science, 38 for math and 16 for English. Rs11264236 in the Natriuretic Peptide Receptor 1 (NPR1) gene and rs10905791 in the Ankyrin Repeat And SOCS Box Containing 13 (ASB13) gene showed suggestive evidence of association with science. The A allele of rs11264236 and the T allele of rs10905791 showed tendencies to increase academic scores in science. In addition, phenotypic correlation analysis revealed that science was significantly more correlated with English and maths than these were with each other.


1691705276393


1691705302475


Academic attainment (broadly defined as performance in educational benchmarks such as national exams and curriculum assessments; AA) consistently and reliably associates with a diverse array of emotional, cognitive and health outcomes1,2. As a consequence, identifying the contributions of specific genetic (and environmental) factors to individual differences in AA and understanding the aetiology of these observed relationships could be informative for a range of societal issues.

A large body of twin and DNA-based research robustly demonstrates that AA and learning abilities are heritable, with a recent meta-analysis of 61 twin studies reporting a heritability estimate of 66% for AA3. The first genome-wide association studies (GWAS) of learning abilities using modest (< 2500) sample sizes demonstrated that they are highly polygenic with thousands of common DNA variants contributing to heritability46. Furthermore, both twin and DNA-based studies suggest that the same genetic variants influence variability in cognitive functions across domains—dubbed ‘generalists genes’7. As a consequence, there has been a shift away from examining finer-grained measures of cognitive processes towards performing population-scale GWAS of broad measures of academic attainment, primarily how long one spends in education (EduYrs). The first EduYrs GWAS (N = 126,559) was a meta-analysis of 42 cohorts performed by the Social Science Genetic Association Consortium (SSGAC; thessgac.org). Three independent genome-wide significant single nucleotide polymorphisms (SNPs) were identified, each accounting for around 0.02% of the variance in EduYrs. Functional interrogation revealed that the signals were located in (or close to) genes previously associated with health and cognition, and demonstrated the utility of taking a broad phenotype to maximise discovery of common genetic variants of very small effect8,9. The subsequent SSGAC GWAS (EduYrs2; N = 293,723) identified a further 74 SNP associations that were mostly located in regions involved in the regulation of gene expression during foetal brain development10. The most recent SSGAC GWAS comprised over 1 million people (EduYrs3) and reported 1271 significantly associated loci and an enrichment of genes involved in neurophysiological brain functions. These included genes involved in neurotransmitter secretion, the activation of ion channels, synaptic plasticity, as well as those expressed in neural tissue both pre- and postnatally11. Construction of an EduYrs genome-wide polygenic score (GPS; a score representing an individual’s amalgamated genetic liability derived from the GWAS results) was predictive of 7–10% of the variance in general cognitive ability and 11–13% in educational attainment, making it one of the most predictive polygenic scores currently available in the behavioural sciences11. The EduYrs GPS associates with many phenotypes, which is perhaps not surprising: such a broad-brush measure taken in adulthood (many levels removed from cell structure and brain function) will necessarily capture a wide range of variables influencing the length of time people stay in formal education. For example, beyond cognitive ability the EduYrs GPS predicts traits such as child behavioural problems, negative symptoms of affect related to depression, callous and unemotional traits, and Attention Deficit Hyperactivity Disorder (ADHD)12,13. If these polygenic scores are to have practical applications, we need a much better understanding of the specificity and generality of genetic causes of variance in educational performance across development.
 
This is known as kindness.
 
This was as autistic as I expected, well done.
 
This was as autistic as I expected, well done.

No. I'm quite certain you enjoyed many and deliberately lied about your history because you were handed holes for looks.

You'll be fine on here.
 
1696952595621


Sim.

Thank you.
 

Exploring the functional role of the CHRM2 gene in human cognition: results from a dense genotyping and brain expression study​


Background

The CHRM2 gene, located on the long arm of chromosome 7 (7q31-35), is involved in neuronal excitability, synaptic plasticity and feedback regulation of acetylcholine release, and has been implicated in higher cognitive processing. The aim of this study is the identification of functional (non)coding variants underlying cognitive phenotypic variation.

Methods

We previously reported an association between polymorphisms in the 5'UTR regions of the CHRM2 gene and intelligence.. However, no functional variants within this area have currently been identified. In order to identify the relevant functional variant(s), we conducted a denser coverage of SNPs, using two independent Dutch cohorts, consisting of a children's sample (N = 371 ss; mean age 12.4) and an adult sample (N= 391 ss; mean age 37.6). For all individuals standardized intelligence measures were available. Subsequently, we investigated genotype-dependent CHRM2 gene expression levels in the brain, to explore putative enhancer/inhibition activity exerted by variants within the muscarinic acetylcholinergic receptor.

Results

Using a test of within-family association two of the previously reported variants – rs2061174, and rs324650 – were again strongly associated with intelligence (P < 0.01). A new SNP (rs2350780) showed a trend towards significance. SNP rs324650, is located within a short interspersed repeat (SINE). Although the function of short interspersed repeats remains contentious, recent research revealed potential functionality of SINE repeats in a gene-regulatory context. Gene-expression levels in post-mortem brain material, however were not dependent on rs324650 genotype.

Conclusion

Using a denser coverage of SNPs in the CHRM2 gene, we confirmed the 5'UTR regions to be most interesting in the context of intelligence, and ruled out other regions of this gene. Although no correlation between genomic variants and gene expression was found, it would be interesting to examine allele-specific effects on CHRM2 transcripts expression in much more detail, for example in relation to transcripts specific halve-life and their relation to LTP and memory.


...

1696953648521

1696953554545

1696953613027


1696953697692

1696953702864

1696953715804

1696953764334

1696953748954
 
It's always felt unnatural to me how much the West prizes loud fast talkers over quiet people who deliver results.
Hitler and Goebbels were great orators and communicators, while Himmler, Albert Speer, and Karl Dönitz were quiet competent people who delivered results.
 

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