1. Description of autistic incel suffering
Many of us suffer because we are autistic incels But suffering can be reduced by treating the underlying causes, and by researching my own condition and reading this forum, I have identified certain patterns. And maybe I find a reason why normal therapy dont work for us.i
In short therapy does not solve our problem and you can’t really explain that to a normie but suffering can be reduced by treating the causes and it is possible. The cause of our suffering is the activity of our brain, which causes additional symptoms that a normee will not experience.
2. Distinguishing neurological Cause from Aggravating Factors
According to my hypothesis, the core cause of our depression like symptoms is not involuntary celibacy itself, but differences in how the autistic brain functions. While the absence of relationships can intensify stress and suffering, it should be understood as an aggravating factor rather than the root cause of the problem.( And this is why normie won't experience such depression from loneliness like us)
3. Proposed Mechanism: Autistic Burnout and HPA Axis Dysregulation
My hypothesis proposes that a significant portion of these difficulties is linked to autistic burnout and dysregulation of the HPA axis (hypothalamic–pituitary–adrenal axis), which is responsible for the body’s stress response and some depression like symptoms(https://pmc.ncbi.nlm.nih.gov/articles/PMC9334910)Chronic overload, prolonged stress, and constant adaptation to a neurotypical environment may lead to deep physical and psychological exhaustion in autistic individuals.
4. Why can it work like this?
In autistic individuals the HPA axis often shows altered stress regulation compared to neurotypical people: stress responses tend to be stronger and longer-lasting, and the diurnal cortisol rhythm is flatter and more irregular (e.g., higher evening cortisol). This pattern can contribute to chronic stress and autistic burnout due to sustained HPA activation.
(Study Corbett et al. – autistic children showed higher cortisol responses to stress and slower recovery vs controls.
Corbett et al. (2024) – autistic youth exhibited a flatter diurnal cortisol slope and elevated evening cortisol, suggesting HPA dysregulation.)
5. Common Symptoms Associated with This State
Symptoms that may indicate autistic burnout and HPA axis dysregulation include:
chronic stress,
low blood pressure,
physical and mental weakness,
sleep disturbances,
brain fog,
substance use as a coping mechanism,
anhedonia (coping strategies no longer provide relief),
functioning on “autopilot,”
memory and concentration problems.
When many of these symptoms occur together, it is possible that they stem from a shared underlying mechanism.
6. Potential Path Toward Improvement
Under this hypothesis, addressing autistic burnout and restoring partial regulation of the HPA axis could allow for a return to basic functioning. This would not reduce our pain from involuntary celibate, but it could significantly reduce suffering and restore a degree of agency, making deliberate and and even for starting small actions against the modern world (of course, the less risky ones that actually have an effect). Obviously, one person won’t change the world, but many people can make a difference—even acting individually. And I’m not talking here about ER, which brings no results and can only cause consequences for us incels.if we want change anything we first have to fix our functioning.
7. Limitations and Conclusion
I want to emphasize that this is my personal hypothesis, based on my own experience and observations of similar patterns in others in this forum. I do not claim that it explains every case or that it constitutes a complete clinical theory. However, identifying a plausible underlying mechanism may be an important first step, as it opens the possibility of seeking targeted treatment, even if full recovery and reducing incel suffering is not guaranteed.
8. From the Author
Since my English is weak and I had to use ChatGPT to translate my text, there may be some errors in the translation. Of course, intensifying awareness of the problem is only the beginning, but I believe that, even if not alone, with a few people here we can actually reduce our suffering, rather than just experiencing a placebo-level effect. If anyone wants, I would be very grateful for help in expanding this text, because at the moment I am in a very weak state and this is all I was able to write.
Many of us suffer because we are autistic incels But suffering can be reduced by treating the underlying causes, and by researching my own condition and reading this forum, I have identified certain patterns. And maybe I find a reason why normal therapy dont work for us.i
In short therapy does not solve our problem and you can’t really explain that to a normie but suffering can be reduced by treating the causes and it is possible. The cause of our suffering is the activity of our brain, which causes additional symptoms that a normee will not experience.
2. Distinguishing neurological Cause from Aggravating Factors
According to my hypothesis, the core cause of our depression like symptoms is not involuntary celibacy itself, but differences in how the autistic brain functions. While the absence of relationships can intensify stress and suffering, it should be understood as an aggravating factor rather than the root cause of the problem.( And this is why normie won't experience such depression from loneliness like us)
3. Proposed Mechanism: Autistic Burnout and HPA Axis Dysregulation
My hypothesis proposes that a significant portion of these difficulties is linked to autistic burnout and dysregulation of the HPA axis (hypothalamic–pituitary–adrenal axis), which is responsible for the body’s stress response and some depression like symptoms(https://pmc.ncbi.nlm.nih.gov/articles/PMC9334910)Chronic overload, prolonged stress, and constant adaptation to a neurotypical environment may lead to deep physical and psychological exhaustion in autistic individuals.
4. Why can it work like this?
In autistic individuals the HPA axis often shows altered stress regulation compared to neurotypical people: stress responses tend to be stronger and longer-lasting, and the diurnal cortisol rhythm is flatter and more irregular (e.g., higher evening cortisol). This pattern can contribute to chronic stress and autistic burnout due to sustained HPA activation.
(Study Corbett et al. – autistic children showed higher cortisol responses to stress and slower recovery vs controls.
Corbett et al. (2024) – autistic youth exhibited a flatter diurnal cortisol slope and elevated evening cortisol, suggesting HPA dysregulation.)
5. Common Symptoms Associated with This State
Symptoms that may indicate autistic burnout and HPA axis dysregulation include:
chronic stress,
low blood pressure,
physical and mental weakness,
sleep disturbances,
brain fog,
substance use as a coping mechanism,
anhedonia (coping strategies no longer provide relief),
functioning on “autopilot,”
memory and concentration problems.
When many of these symptoms occur together, it is possible that they stem from a shared underlying mechanism.
6. Potential Path Toward Improvement
Under this hypothesis, addressing autistic burnout and restoring partial regulation of the HPA axis could allow for a return to basic functioning. This would not reduce our pain from involuntary celibate, but it could significantly reduce suffering and restore a degree of agency, making deliberate and and even for starting small actions against the modern world (of course, the less risky ones that actually have an effect). Obviously, one person won’t change the world, but many people can make a difference—even acting individually. And I’m not talking here about ER, which brings no results and can only cause consequences for us incels.if we want change anything we first have to fix our functioning.
7. Limitations and Conclusion
I want to emphasize that this is my personal hypothesis, based on my own experience and observations of similar patterns in others in this forum. I do not claim that it explains every case or that it constitutes a complete clinical theory. However, identifying a plausible underlying mechanism may be an important first step, as it opens the possibility of seeking targeted treatment, even if full recovery and reducing incel suffering is not guaranteed.
8. From the Author
Since my English is weak and I had to use ChatGPT to translate my text, there may be some errors in the translation. Of course, intensifying awareness of the problem is only the beginning, but I believe that, even if not alone, with a few people here we can actually reduce our suffering, rather than just experiencing a placebo-level effect. If anyone wants, I would be very grateful for help in expanding this text, because at the moment I am in a very weak state and this is all I was able to write.
